Kurum Yazarı "Azık, Fatih Mehmet" İçin Listeleme
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beta 2-Microglobulin, Neutrophil Gelatinase-Associated Lipocalin, and Endocan Values in Evaluating Renal Functions in Patients with beta-Thalassemia Major
Çetinkaya, Petek Uzay; Azık, Fatih Mehmet; Karakuş, Volkan; Huddam, Bülent; Yılmaz, Nigar (Taylor & Francis Ltd, 2020)Chronic anemia, transfusion-associated iron deposition, and chelating agents lead to renal impairment in beta-thalassemia (beta-thal) patients. The present study aimed to determine the most reliable and practical method ... -
Difficulties in the diagnosis of Hb S/Beta thalassemia: Really a mild disease?
Uçucu, Süheyl; Karabıyık, Talha; Azık, Fatih Mehmet (SOC MEDICAL BIOCHEMISTS SERBIA, 2022)Background: HbS/beta cases having clinical, hematologic and electrophoretic similarities cannot be sufficiently distinguished from sickle cell anemia cases and are misdiagnosed as sickle cell anemia. This study will ... -
The effect of measurement area size on the reliability of myocardial iron load measurement in cardiac magnetic resonance imaging examinations of thalassemia patients
Yeniçeri, İbrahim Önder; Azık, Fatih Mehmet; Elibol, Funda Dinç (Turkiye Klinikleri, 2021)Background/aim: The aim of this study was to evaluate the intraobserver and interobserver reliability of cardiac T2* MRI measurements in different region of interest (ROI) sizes. Materials and methods: Cardiac T2* MRIs of ... -
IVS-II-16 (G>C) (HBB: c.315+16G>C) or IVS-II-666 (C>T) (HBB: c.316-185C>T) Mutations Trigger an Hb S (HBB: c.20A>T)/β+-Thalassemia Phenotype in an Hb S Trait Patient
Uçucu, Süheyl; Karabıyık, Talha; Azık, Fatih Mehmet (Taylor and Francis Ltd., 2021)Sickle cell trait is a medical condition caused by the presence of both mutant Hb S (HBB: c.20A>T) and normal Hb A alleles. Although sickle cell trait is typically considered to be asymptomatic and benign, genetic modifiers ...